ИЗУЧЕНИЕ КЛИНИЧЕСКОГО СЛУЧАЯ. ТЯЖЁЛАЯ ГИПОНАТРИЕМИЯ С ВЫСОКИМ СОДЕРЖАНИЕМ НАТРИЯ В МОЧЕ И ОСМОЛЯЛЬНОСТЬЮ

первичная недостаточность коры надпочечников, синдром неадекватной секреции антидиуретического гормона (СНСАДГ), гипонатриемия, осмоляльность

1. Hoorn EJ, Zietse R. Hyponatremia revisited: translating physiology to practice. Nephron Physiol 2008; 108: 46-59

2. Lin SH, Hsu YJ, Chiu JS, Chu SJ, Davids MR, Halperin ML. Osmotic demyelination syndrome: a potentially avoidable disaster. QJM 2003; 96: 935-947

3. Fenske W, Stцrk S, Blechschmidt A, Maier SG, Morgenthaler NG, Allolio B. Copeptin in the differential diagnosis of hyponatremia. J Clin Endocrinol Metab 2009; 94: 123-129

4. Decaux G, Musch W. Clinical laboratory evaluation of the syndrome of inappropriate secretion of antidiuretic hormone. Clin J Am Soc Nephrol 2008;3:1175-1184

5. Smith JC, Siddique H, Corrall RJ. Misinterpretation of serum cortisol in a patient with hyponatraemia. BMJ 2004; 328: 215-216

6. Soule S. Addison’s disease in Africa: a teaching hospital experience. Clin Endocrinol (Oxf) 1999;50:115-120

7. Pura M, Kreze Jr A, Kento P, Vauga P. The low-dose (1 microg) cosyntropin test (LDT) for primary adrenocortical insufficiency: defining the normal cortisol response and report on first patients with Addison disease confirmed with LDT. Exp Clin Endocrinol Diabetes 2010;118(3):151-157

8. Cherney DZ, Davids MR, Halperin ML. Acute hyponatraemia and «ecstasy»: insights from a quantitative and integrative analysis. QJM 2002;95:475-483

9. Gagnon RF, Halperin ML. Possible mechanisms to explain the absence of hyperkalaemia in Addison’s disease. Nephrol Dial Transplant 2001;16:1280-1284

10. Quinn SJ, Kifor O, Trivedi S, Diaz R, Vassilev P, Brown E. Sodium and ionic strength sensing by the calcium receptor. J Biol Chem 1998; 273:19579-19586